杤尾 人司¹, 冨田 周介², 工藤 正俊², 岡部 純弘², 樫田 博史², 岩崎 信広¹, 簑輪 和士¹, 曽我 登志子¹, 田村 周二¹, 森本 義人¹, 渡辺 智裕², 福永 豊和², 平佐 昌弘², 伊吹 康良², 織野 彬雄², 藤堂 彰男²

Hitoshi TOCHIO¹, Syusuke TOMITA², Masatoshi KUDO², Yoshihiro OKABE², Hiroshi KASHIDA², Nobuhiro IWASAKI¹, Kazushi MINOWA¹, Toshiko SOGA¹, Syuji TAMURA¹, Yoshito MORIMOTO¹, Tomohiro WATANABE², Toyokazu FUKUNAGA², Masahiro HIRASA², Yasuyosi IBUKI², Akio ORINO², Akio TODA²

¹神戸市立中央市民病院臨床病理科腹部超音波検査室, ²神戸市立中央市民病院消化器センター内科

¹Division of Abdominal Ultrasound, Kobe City General Hospital, ²Department of Gastroenterology, Kobe City General Hospital

キーワード : Adenomatous hyperplasia, Chronic liver disease, Color Doppler imaging, Continuous wave form signal, Well-differentiated HCC

We evaluated size, hemodynamic pattern, and histologic findings of 26 nodules associated with liver cirrhosis from which color Doppler imaging obtained afferent continuous wave form signals. Nodule size ranged from 0.8 to 3.0 cm (2.1±0. 6 cm (SD)). Arterial vascularity determined by US angiography was hypovascular, 16 noeules; vascular spot in hypovascular, 7 nodules; and isovascular, 3 nodules. No hypervascular nodules were observed. CT conducted during arterial portgraphy showed portal supply in 25 (96%) of the 26 nodules. Slightly increased portal perfusion was observed in 6 nodules; slightly decreased portalperfusion, in 5 nodules. Portal per fusion in the remaining 14 nodules was identical to that of the surrounding liver parenchyma. partial portal per fusion defects were observed in the 2 cases with increased portal per fusion and in 1 case with identical portal per fusion. Histologic diagnosis showed 12 cases to be hyperplastic nodules and 14 cases to be well-differentiated HCC. No moderately or poorly differentiated HCC was obsened. we conclude that nodules that produced afferent intratumoral continuous wave form signals are either hyperplastic nodules or well-differentiated HCCs, not typical, classical HCC